Anesthesia Emergencies comprises correct step by step details on find out how to become aware of, deal with, and deal with problems and emergencies through the perioperative interval. Concisely written, highlighted sections on fast administration and hazard components make stronger crucial issues for simple memorization, whereas constant association and checklists offer ease of studying and readability. Anesthesia prone will locate this e-book an imperative source, describing overview and therapy of life-threatening events, together with airway, thoracic, surgical, pediatric, and cardiovascular emergencies. the second one version includes a revised desk of contents which offers themes so as in their precedence in the course of emergencies, in addition to new chapters on predicament source administration and catastrophe medicine.
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Additional info for Anesthesia Emergencies (2nd Edition)
The abscess may rupture spontaneously or after attempts at laryngoscopy and intubation, flooding the hypopharynx with pus that may then be aspirated. Further Reading Boscolo-Rizzo P, Da Mosto MC. Submandibular space infection: a potentially lethal infection. Int J Infect Dis. 2009; 3(3): 327–333. 31 Anesthesia Emergencies 32 Marple BF. Ludwig angina: a review of current airway management. Arch Otolaryngol Head Neck Surg. 999; 25(5): 596–599. Ovassapian A, Tuncbilek M, Weitzel EK, Joshi CW.
Pathophysiology The early phase of ARDS is characterized by pulmonary capillary leak and interstitial and alveolar edema. There is a loss of surfactant activity. During the late phase of ARDS, pulmonary fibrosis and decreased lung compliance can develop. Immediate Management • Increase FIO2 and titrate positive end-expiratory pressure (PEEP) to maintain adequate oxygenation. • Consider ventilation with low tidal volumes (4–8 cc/kg predicted body weight). • Avoid plateau pressures >30 cm H2O. • Permissive hypercapnia may be necessary.
Poor lung compliance may reduce minute ventilation and cause hypercarbia. Residual anesthetic effects or inadequate reversal of muscle relaxants can cause postoperative hypercarbia. Splinting due to pain can lead to increased dead space, hypoventilation, and hypercarbia. Hypermetabolic states and fever may contribute to increased CO2 production. Immediate Management • Intubate the trachea and initiate mechanical ventilation for severe respiratory acidosis, if the patient is unable to protect his or her airway, or if respiratory failure is imminent.