By David A. Clark PhD, Aaron T. Beck MD
Updating and reformulating Aaron T. Beck’s pioneering cognitive version of hysteria problems, this groundbreaking paintings is either authoritative and hugely useful. The authors synthesize the most recent pondering and empirical info on anxiousness remedy and provide step by step guideline in cognitive overview, case formula, cognitive restructuring, and behavioral intervention. they supply evidence-based mini-manuals for treating the 5 commonest nervousness problems: panic affliction, social phobia, generalized nervousness disease, obsessive–compulsive illness, and posttraumatic rigidity ailment. easy good points comprise vibrant case examples, concise “Clinician instructions” that strengthen key issues, and over 3 dozen reproducible handouts and varieties. (20100401)
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Extra info for Cognitive Therapy of Anxiety Disorders: Science and Practice
Although this primal behavioral mobilization evolved as a rapid and efficient response to physical danger, it can impair actual performance when activated in benign situations or the complex, diffusely stressful circumstances of contemporary society. , 1985; D. M. Clark & Beck, 1988). , 1985). ” It is associated with the cognitive perspective of being totally helpless. The immobility response is apparent in social anxiety, such as when a highly anxious person feels faint when attempting to deliver a public speech.
The same holds true for public speaking. If you evaluate your audience as friendly and receptive to your speech, your anxiety will be lower than if you evaluate the audience as critical, bored, or rejecting of your talk. , writing an exam, giving a speech, or having a casual conversation) that determines the level of anxiety, but rather how the situation is appraised or evaluated. It is the way we think that has a powerful influence on whether we feel anxious or calm. The cognitive perspective can help us understand some apparent contradictions in anxiety disorders.
The CRH, then, not only mediates endocrine responses to stress but also other broad brain and behavioral responses that play a role in the expression of stress, anxiety, and depression (Barlow, 2002). Overall, then, abnormalities at the neurotransmitter level appear to have anxiogenic or anxiolytic effects that play an important contributory role in heightened physiological states that characterize fear and anxiety. However, the exact nature of these abnormalities is still unknown. 4 provides a summary of the biological aspects of anxiety that might underlie the cognitive features of these disorders discussed later in this volume.